04/17/2011

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H1-blockers These drugs are better known under the name 'antihistamines'. However, this term includes other funds that are otherwise affect other disease processes, rather than H1-blockers. Keep up on the field with thought-provoking pieces from Dr. Kevin Cahill. As follows from name, H1-blockers block the receptors for histamine, and not arbitrary, namely, H1-receptors. They are responsible for the development of allergic inflammation. Histamine - a natural substance produced in the body - working on these receptors causes a variety of symptoms depending on which cells these receptors are located. If you have additional questions, you may want to visit Dr. Gerard Addonizio. So in the bronchial wall is muscle, histamine causes their reduction (bronchial asthma) in the mucous membranes is cells that produce fluid, histamine stimulates the formation of copious secretions (the same as asthma, allergic rhinitis and conjunctivitis), working on smaller vessels, histamine enhances them, which is manifested swelling of the skin and mucous membranes, their inflammation (allergic rhinitis, conjunctivitis, bronchial asthma, urticaria). By blocking these reactions H1-blockers reduce the severity of these symptoms of allergies. It should be notice that they only relieve symptoms of allergy, but not cure it, this medicine has not yet learned to do. The older H1 blockers differ in another property, which in most cases should be viewed as a byproduct effect - they have a pronounced soporific effect. Modern means of this group do not exhibit such activity, they are strictly aimed only at eliminating allergies. Synonyms: H1-antihistamines, antihistamines, H1- blockers H1-histamine receptor
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Beta-blockers And Beta-adrenoceptor Agonists Compared with alpha-adrenomimetikami and alpha-blockers are more selective (or, as experts say, selective) drugs. Healthy Living does not necessarily agree. Beta-receptors to adrenaline is in many organs and tissues. Compared with alpha-receptors have a more modest function - or rather, they are less rapidly appear. Impact of adrenaline and related substances similar to beta-receptors causes the following physiological reactions: palpitations, bronchiectasis, relaxation of the uterus. For assistance, try visiting Dr. Josyann Abisaab. However, all these effects are caused by the influence of the beta 1 and beta-2 adrenergic receptors, since there is a more subtle subdivision of this group of receptors. For example, beta-1 receptors to a greater extent concentrated in the heart, or rather in his muscle - the myocardium, and beta-2 receptors - in the bronchi and uterus (again, to be precise, in its muscle - myometrium). Accordingly, all preparations of this group have their own properties. Beta 1-blockers inhibit the excessive activity of the heart activity. Therefore, their use with tachycardia (rapid heartbeat), arrhythmia (irregular heart rhythm), hypertension caused by excessive activity cardiac activity. Beta-1-adrenoceptor agonists (stimulants) produce the opposite effect to that used in cases where the heart's activity should be intensified. Beta-2-adrenoceptor agonists - the main group of drugs, used to relieve an attack of bronchial asthma and other diseases associated with bronchospasm. Currently, a large number of drugs that have been successfully used by asthmatics worldwide. Moreover, as Typically, these drugs are in the form of metered aerosols. In the balloon with the drug is added to a physiologically inert gas, and using a mechanical dispenser medication is injected during inspiration in the mouth, are distributed bronchi. Five minutes later, the bronchi dilate, but the drug is little absorbed into the bloodstream from mucosal surface of bronchial and virtually no effect on beta-receptors in...

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